Magnetic resonance findings in subacute combined degeneration

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Magnetic resonance findings in subacute combined degeneration.

2. Hathout L, El-Saden S. Nitrous oxide-induced B12 deficiency myelopathy: perspectives on the clinical biochemistry of vitamin B12. J Neurol Sci. 2011;301(1-2):1-8. https://doi.org/10.1016/j.jns.2010.10.033 3. Gupta PK, Gupta RK, Garg RK, Rai Y, Roy B, Pandey CM et al. DTI correlates of cognition in conventional MRI of normal-appearing brain in patients with clinical features of subacute combi...

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Subacute combined degeneration: clinical, electrophysiological, and magnetic resonance imaging findings.

OBJECTIVE Vitamin B12 deficiency is a systemic disease that often affects the nervous system. One of the most prevalent manifestations is subacute combined degeneration (SCD) of the spinal cord. To access the clinical, electrophysiological, and structural abnormalities associated with SCD, a study was conducted in nine patients. METHODS Clinical, electrophysiological (electroneurography, soma...

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Two cases of subacute combined degeneration: magnetic resonance findings.

We report two cases of subacute combined degeneration. Both patients had undergone total gastrectomy. The chief complaints were numbness in both upper extremities in case 1 and numbness in both the upper and lower extremities and gait disturbance in case 2. The pain, temperature, and vibration senses of both patients were decreased. Laboratory examinations showed macrocytic anemia and a decreas...

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Subacute combined degeneration : report of two cases

Subacute combined degeneration ( SCD) due to vitamin B12 deficiency is common in North of Europe but sometimes it is seen in Iran. It can develop before the symptoms of anemia is obvious, with myelin injury and then axon and even neuron destruction in spinal cord, brain, optic and peripheral nerves. In this study two cases are reported; in the first patient, this disease in manifested by vitami...

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ژورنال

عنوان ژورنال: Arquivos de Neuro-Psiquiatria

سال: 2017

ISSN: 1678-4227,0004-282X

DOI: 10.1590/0004-282x20170068